In simple terms
A friendly intro before the formal notes — no formulas yet.
Why the Body Breaks Down Under Pressure
A health problem like stress or addiction is never just 'a body thing'. Biology sets up the machinery (hormones, dopamine), the mind decides how to read the situation (appraisal, expectancy), and the social world supplies the triggers (a low-control job, drug-using peers). A good essay explains one problem through more than one of these lenses and backs each with a study.
Think of stress like a car alarm. Biology is the alarm system wired into the car (the HPA axis) — useful in an emergency, damaging if it blares for months. Cognition is the sensitivity dial: two identical cars can have the alarm set to go off at a gentle knock or only at a real break-in — that dial is your appraisal of the threat. The social world is the neighbourhood: park on a rough street (a low-control, high-demand job) and the alarm is triggered far more often. To explain why one car's battery keeps dying, you need all three.
- 1
Choose ONE health problem (stress or addiction) and stick to it across the whole essay.
- 2
Explain it from at least two perspectives — e.g. biological (HPA/cortisol) and cognitive (appraisal).
- 3
For each explanation, USE one study: state the finding and link it straight back to the explanation and the question.
- 4
Evaluate — reductionism, correlation vs causation, and how the perspectives combine in the biopsychosocial model — then conclude.
Explore the concept
Use the live diagram and synced steps — play it or tap a step card to walk through.
Full topic notes
Formal explanation with the rigour you need for the exam.
How examiners frame 'health problems'
Health psychology rests on the biopsychosocial model: health and illness emerge from the interaction of biological factors (genes, hormones, neurotransmitters), psychological factors (thoughts, appraisals, expectancies) and social factors (culture, socioeconomic status, relationships). For any single health problem, all three levels are relevant, and a strong essay explains ONE problem through at least two of them rather than listing symptoms.
Watch the command term. 'Explain' or 'Outline' wants the mechanism made clear; 'Discuss' or 'Evaluate' additionally wants critical thinking — strengths, limitations and competing perspectives. Whatever the term, the structural move is the same: choose one problem, commit to it, and support each explanation with a study you actually use.
Health problem in focus: stress
Stress is the body's response to a stressor — any demand perceived as threatening or exceeding one's resources. Acute stress is adaptive: it sharpens attention and mobilises energy to deal with an immediate challenge. The problem is chronic stress. When the response stays switched on for weeks or months, the same systems that once protected us begin to cause harm: chronic stress is associated with hypertension and cardiovascular disease, suppressed immune function, impaired memory, and raised risk of anxiety and depression. Because prolonged activation is so common in modern life, stress is one of the most widely studied — and most examinable — health problems in the option.
Biological explanation: the HPA axis and cortisol
The physiological stress response runs on two pathways. The FAST one is the SAM axis (sympathetic-adrenal-medullary): the sympathetic nervous system signals the adrenal medulla to release adrenaline within seconds, producing the racing heart and heightened alertness of 'fight or flight'. The SLOWER, sustained one is the HPA axis (hypothalamic-pituitary-adrenal): the hypothalamus releases CRH, which prompts the pituitary to release ACTH, which drives the adrenal cortex to release cortisol. Cortisol frees glucose for prolonged effort — useful in the short term. But when cortisol stays elevated for months, it is implicated in damage to the hippocampus (harming memory), suppression of the immune system, and cardiovascular strain. This is the mechanism by which a psychological experience becomes a physical illness.
Supporting study — Newcomer et al. (1999). Aim: to test whether cortisol at the levels produced during stress impairs memory. Procedure: healthy adults were given tablets over several days — a high dose of cortisol equivalent to a major stressor, a low dose, or a placebo — and were tested on verbal declarative memory (recalling prose). Findings: the high-cortisol group showed significantly worse recall than the low-dose and placebo groups, and the impairment reversed once the cortisol was stopped. USING this study: it provides experimental (not merely correlational) support for the HPA explanation — manipulating cortisol directly changed cognitive performance — which is why we can argue cortisol is a cause of stress's harmful effects on memory, not just a correlate.
SAM axis — fast: sympathetic nervous system -> adrenal medulla -> ADRENALINE. Seconds. Racing heart, 'fight or flight'.
HPA axis — slow, sustained: hypothalamus (CRH) -> pituitary (ACTH) -> adrenal cortex -> CORTISOL. Minutes to hours; can stay elevated for months.
Chronic cortisol harms: hippocampal/memory damage, immune suppression, raised cardiovascular risk — the bridge from psychological stress to physical illness.
Key discrimination: adrenaline is SAM; cortisol is HPA. Mixing them up is a recurring examiner-penalised error.
Cognitive explanation: appraisal (Lazarus & Folkman)
The biological account explains the machinery but not why the same event floors one person and energises another. Lazarus and Folkman's transactional model (1984) fills that gap. Stress, they argue, arises from the transaction between the person and the situation, mediated by two appraisals. PRIMARY appraisal asks 'Is this a threat, a challenge, or irrelevant?'. SECONDARY appraisal asks 'Do I have the resources to cope?'. Stress results only when perceived demands exceed perceived coping resources. On this view it is not the event itself but its interpretation that triggers the stress response — which is precisely why the model can explain individual differences that biology alone cannot.
Supporting study — Speisman et al. (1964). Aim: to test whether manipulating cognitive appraisal changes the physiological stress response to the SAME stimulus. Procedure: participants watched a distressing film of a painful rite while hearing one of several soundtracks — a 'trauma' track emphasising the pain, a 'denial' track downplaying it, an 'intellectualisation' track giving a detached anthropological narration, or silence. Physiological arousal (heart rate, galvanic skin response) was recorded. Findings: arousal was highest with the trauma track and markedly lower with the denial and intellectualisation tracks, even though the images were identical. USING this study: because only the appraisal — not the stimulus — was varied, the result directly supports the claim that appraisal shapes the stress response, giving the cognitive explanation experimental grounding.
Social factors in stress
Stress is not only inside the body and mind; social conditions shape how often the response fires and how long it lasts. Chronic stressors cluster by socioeconomic status: financial insecurity, poor housing, discrimination and, in particular, low control over one's work. The Whitehall studies of British civil servants (Marmot and colleagues) found a striking social gradient: lower employment grades had substantially higher rates of stress-related cardiovascular disease, and this was linked especially to low job control — how much say a person had over their own work — rather than to income alone. USING this: it shows a specifiable social variable (job control) predicts a physical health outcome, evidencing the sociocultural explanation and demonstrating that stress is patterned by social structure, not just individual temperament.
The alternative: explaining addiction
If you prefer addiction as your 'one health problem', the demands are identical — explain it from more than one perspective, using research. Addiction is a chronic, relapsing condition of compulsive drug use (or behaviour) despite harm, with tolerance and withdrawal. It carries heavy effects — health damage, social and economic cost — and high relapse rates, which is exactly why single-perspective explanations fall short.
Biological — dopamine reward: addictive drugs sharply raise dopamine in the mesolimbic pathway (VTA -> nucleus accumbens), powerfully reinforcing use. Volkow et al.'s neuroimaging work links addiction to reduced dopamine D2 receptor availability, so more drug is needed for the same reward (tolerance) — a mechanism for compulsive use. USE it to argue addiction has a neurobiological basis, not just weak willpower.
Cognitive — expectancy and attentional bias: users hold outcome expectancies ('this will relax me / help me cope') that drive use, and develop an attentional bias toward drug cues. Field and Cox's review of attentional-bias research shows addicted individuals' attention is automatically captured by drug-related stimuli, feeding craving and relapse — evidence for a cognitive maintenance mechanism.
Sociocultural — social learning: Bandura's social learning theory explains initiation through observation and imitation of models (peers, family, media) and vicarious reinforcement — seeing use rewarded with status or belonging. Consistent findings that parental and peer substance use predict adolescent uptake support a social pathway into addiction.
Evaluating explanations of health problems
Reductionism vs holism: a purely biological account (cortisol, dopamine) explains mechanism but ignores meaning and context; a purely social account ignores why exposed individuals differ. The biopsychosocial model integrates them — say so explicitly, and use one perspective to critique another.
Correlation vs causation: much health-psychology evidence is correlational (e.g. Whitehall's link between job control and heart disease). Prefer 'is associated with' / 'is a risk factor for'. Praise experimental studies (Newcomer; Speisman) precisely because manipulation licenses causal claims.
Individual and cultural differences: appraisal and coping vary across people and cultures, and much research (including the Whitehall sample) is drawn from specific populations — so limits generalisability.
Ecological validity vs control: lab studies (Speisman's film) gain control but may not capture real chronic stress; field/epidemiological studies (Whitehall) gain realism but lose control. Use this trade-off to weigh evidence, not just list it.
Bidirectionality: stress and health, or drug use and D2 receptors, may influence each other in both directions — a mature point that lifts Criterion D.
Common mistakes examiners penalise
Confusing SAM and HPA: writing that the HPA axis releases adrenaline, or that cortisol is the fast 'fight or flight' hormone. Adrenaline = SAM (fast); cortisol = HPA (slow). This directly costs Criterion B.
Describing studies instead of USING them: narrating Newcomer's or Speisman's procedure without the sentence that links the finding to the explanation and the question. Under our markband, described-but-not-used research is capped in Criterion C.
Answering on two health problems: the question says 'one'. Covering both stress and addiction sacrifices the depth (Criterion B) and focus (Criterion A) the top bands require.
Treating appraisal as biological, or the HPA axis as cognitive: keep the perspectives straight — appraisal is a COGNITIVE process; the HPA axis is a BIOLOGICAL mechanism.
Claiming causation from correlational data: e.g. asserting low job control 'causes' heart disease from Whitehall alone. Use cautious language and reserve causal claims for experiments.
Evaluation bolted on as a list: strengths/limitations dumped in a final paragraph with no link to the argument. Weave critical thinking through the essay for Criterion D.
Common to SL and HL
The health psychology option, including the 'health problems' content, is common to SL and HL at this level — the explanations, studies and 22-mark Paper 2 essay demands are the same for both. There is no separate HL extension for this material, so the model answer and markband above apply to every student taking this option.
Worked examples
See the formulas applied — reveal one step at a time, like the exam.
Short-answer / SAQ practice: Explain one biological explanation of stress. [9]
- 1
Point: One biological explanation locates the harm of stress in prolonged activation of the HPA axis and the hormone cortisol.
ERQ practice: Explain one biological explanation of addiction, using research. [Discuss-style paragraph]
- 1
Claim: A leading biological explanation is that addiction hijacks the brain's dopamine reward system.
Paper 2 ERQ: Discuss explanations for one health problem (e.g. stress or addiction). [22]
- 1
Model essay (health problem: stress)
How it all connects
The big idea sits in the middle — tap a linked idea to explore the link.
Tap a linked idea to see how it connects back to the main topic — that connection is what examiners reward.
Glossary
Try to recall each definition before you reveal it.
Quick check
Answer in your head first — then tap to check. No pressure.
Revision flashcards
Flip the card. Test yourself before the exam.
Health problem (IB sense)
A physical or mental condition — the option names stress, obesity, chronic pain and addiction — that psychologists explain using biological, cognitive and sociocultural factors. In an essay you usually explain just ONE.
Key takeaways
Review these before you close the topic — retrieval beats re-reading.
- ✓
SAM axis — fast: sympathetic nervous system -> adrenal medulla -> ADRENALINE. Seconds. Racing heart, 'fight or flight'.
- ✓
HPA axis — slow, sustained: hypothalamus (CRH) -> pituitary (ACTH) -> adrenal cortex -> CORTISOL. Minutes to hours; can stay elevated for months.
- ✓
Chronic cortisol harms: hippocampal/memory damage, immune suppression, raised cardiovascular risk — the bridge from psychological stress to physical illness.
- ✓
Key discrimination: adrenaline is SAM; cortisol is HPA. Mixing them up is a recurring examiner-penalised error.
Practice — then mark it
The whole point: a real Cambridge question, marked mark-by-mark.
Get a Paper 2 essay marked: discuss explanations for one health problem (stress or addiction) [22]
Get a Paper 2 essay marked: discuss explanations for one health problem (stress or addiction) [22]
Extra simulations & links
PhET, GeoGebra and other curated tools — open in a new tab.
Frequently asked
Checkpoint
One marked question is worth ten re-reads — close the loop before you move on.
Reading it isn’t knowing it — prove it.
Before you move on: do Get a Paper 2 essay marked: discuss explanations for one health problem (stress or addiction) [22] on paper, snap a photo, and get examiner-style feedback on exactly where you win and lose marks.